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Publisher: Elsevier (Cell Press)
Languages: English
Types: Article
Subjects:

Classified by OpenAIRE into

mesheuropmc: hemic and lymphatic diseases
Effective differentiation therapy for acute myeloid leukemia (AML) has been restricted to a small subset of patients with one defined genetic abnormality. Using an unbiased small molecule screen, Sykes et al. now identify a mechanism of de-repression of differentiation in several models of AML driven by distinct genetic drivers.
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